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Blood
Distinct functional programming of human fetal and adult monocytes.
Response: Maraviroc intensification and microbial translocation.
The immunologic effects of maraviroc intensification in treated HIV-infected individuals with incomplete CD4+ T-cell recovery: a randomized trial.
Activation, exhaustion, and persistent decline of the antimicrobial MR1-restricted MAIT-cell population in chronic HIV-1 infection.
Leaky gut, clotting, and vasculopathy in SIV.
Blood T-cell receptor diversity decreases during the course of HIV infection, but the potential for a diverse repertoire persists.
Evidence for both innate and acquired mechanisms of protection from Plasmodium falciparum in children with sickle cell trait.
Expansion of CD8+ T cells lacking Sema4D/CD100 during HIV-1 infection identifies a subset of T cells with decreased functional capacity.
HIV disease progression despite suppression of viral replication is associated with exhaustion of lymphopoiesis.
HIV disease progression correlates with the generation of dysfunctional naive CD8(low) T cells.
